mir-14 microRNA precursor family
In molecular biology mir-14 microRNA is a short RNA molecule. MicroRNAs function to regulate the expression levels of other genes by several mechanisms.
| mir-14 | |
|---|---|
| Identifiers | |
| Symbol | mir-14 |
| Rfam | RF00752 |
| miRBase family | 7 |
| Other data | |
| RNA type | microRNA |
| Domain(s) | Eukaryota; |
| PDB structures | PDBe |
See also
References
Further reading
- Gao L, Zuo H, Liu K, Li H, Zhong G (2012). "A new strategy for identification of highly conserved microRNAs in non-model insect, Spodoptera litura". International Journal of Molecular Sciences. 13 (1): 612–27. doi:10.3390/ijms13010612. PMC 3269709. PMID 22312275.
- Varghese J, Lim SF, Cohen SM (December 2010). "Drosophila miR-14 regulates insulin production and metabolism through its target, sugarbabe". Genes & Development. 24 (24): 2748–53. doi:10.1101/gad.1995910. PMC 3003191. PMID 21159815.
- Kumarswamy R, Chandna S (August 2010). "Inhibition of microRNA-14 contributes to actinomycin-D-induced apoptosis in the Sf9 insect cell line". Cell Biology International. 34 (8): 851–7. doi:10.1042/CBI20100035. PMID 20486901.
- Mead EA, Tu Z (May 2008). "Cloning, characterization, and expression of microRNAs from the Asian malaria mosquito, Anopheles stephensi". BMC Genomics. 9: 244. doi:10.1186/1471-2164-9-244. PMC 2430712. PMID 18500992.
- Guo L, Ding ZH (October 2007). "[Advances in the studies of miRNAs and cell apoptosis]". Sheng Li Ke Xue Jin Zhan [Progress in Physiology] (in Chinese). 38 (4): 331–5. PMID 18232304.
- Varghese J, Cohen SM (September 2007). "microRNA miR-14 acts to modulate a positive autoregulatory loop controlling steroid hormone signaling in Drosophila". Genes & Development. 21 (18): 2277–82. doi:10.1101/gad.439807. PMC 1973141. PMID 17761811.
- Xu P, Vernooy SY, Guo M, Hay BA (April 2003). "The Drosophila microRNA Mir-14 suppresses cell death and is required for normal fat metabolism" (PDF). Current Biology. 13 (9): 790–5. doi:10.1016/S0960-9822(03)00250-1. PMID 12725740. S2CID 6391484.
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